DISCUSSION
This patient experienced cardiogenic shock due to pheochromocytoma crisis. Early administration of ECMO treatment in the local hospital with other subsequent treatments were crucial in saving the patient’s life and improving their survival rate. Unfortunately, ECMO had to be discontinued because of secondary severe bloodstream infection, possibly caused by emergency catheterization. To address this, CRRT was used as it has demonstrated effectiveness in removing toxins and inflammatory mediators, regulating internal environment and improving circulatory function in cases of multiple traumas and acute kidney injury5. Given the high technical requirements, cost, and patient limitation associated with ECMO, CRRT was continued even after the patient was transferred to our hospital. The results in Table 1 show that prolonged CRRT treatment gradually decreased norepinephrine level in the patient’s body. In this case report, CRRT effectively eliminated catecholamines metabolites. However, the patient developed sepsis, along with toxins and inflammatory cytokines in the circulation. During the acute attack, the patient experienced heart, liver, and kidney damaged, leading to circulatory failure, hemodynamic fluctuations, and inflammatory cytokine secretion. Table 2-3 reveals a rapid onset of acid-base imbalance in the internal environment. After the CRRT treatment, the patient’s serum sodium, potassium, electrolyte ions, pH, as well as and heart, liver, and kidney function gradually improved. Those improvements may be attributed to the removal of toxins, inhibition of the patient’s inflammatory response, and gradually elimination of catecholamines, related substances, and their metabolites.