1-Introduction:
HH is an autosomal recessive disease with low penetrance [1]. The
most common variant is the HFE C282Y mutation. The diagnosis of
HH is established by an elevated ferritin level and a high iron
saturation along with homozygosity for C282Y. Hemochromatosis is a state
of iron-overload. Patients with iron overload are susceptible to
infections. For instance, Yersinia enterocolitica is a
ferrophilic organism that thrives in an environment with an excess of
iron [1]. We herein present three case reports whereby HH was
unveiled by a preceding Yersinia enterocolitica infection in the
setting of elevated iron tissue deposition markers.