HH is an autosomal recessive disease with low penetrance [1]. The most common variant is the HFE C282Y mutation. The diagnosis of HH is established by an elevated ferritin level and a high iron saturation along with homozygosity for C282Y. Hemochromatosis is a state of iron-overload. Patients with iron overload are susceptible to infections. For instance, Yersinia enterocolitica is a ferrophilic organism that thrives in an environment with an excess of iron [1]. We herein present three case reports whereby HH was unveiled by a preceding Yersinia enterocolitica infection in the setting of elevated iron tissue deposition markers.