Multiple myeloma is characterized by the clonal proliferation of malignant plasma cells in the bone marrow, accompanied by the excessive production of monoclonal immunoglobulins or light chains (M protein)[1, 2]. The malignant proliferation of plasma cells and abnormal levels of immunoglobulins lead to clinical symptoms characterized by CRAB, namely C: hypercalcemia, R: renal impairment, A: anemia, and B: bone pain, skeletal changes, or pathological fractures[3]. Depending on the type of immunoglobulin secreted by the abnormal myeloma cells, MM can be classified into eight types: IgG, IgA, IgM, IgD, IgE, biclonal, non-secretory, and light chain (κ, λ)[4, 5].

The majority of MM patients exhibit normal or decreased blood lipid levels. However, isolated cases have reported the significant role of M protein in elevated lipid levels[6-8]. Among these cases, IgA type multiple myeloma is particularly associated with hypertriglyceridemia and hypercholesterolemia.